1、 Duval C, Smit PM, Kolb AM, et al. Keratinocytes control the pheo/eumelanin ratio in cultured normal human melanocytes.Pigment Cell Res,2002,15:440-446. 2、 中国中西医结合学会皮肤性病专业委员会色素病学组.白癜风临床分型与疗效标准 (2003年修订稿).中华皮肤科杂志,2004,37(7):440。 3、 Shimizu M,Saitoh Y,Itoh H.Immunohistochemical staining of Ha-ras oncogene in normal,benign and malignant human pancreatic tissues.Hum Pathol,1990,21(6):607-612. 4、 Moellmann G., Klein-Angere S., Scollay D.A. et al. Extracellular granular material and degeneration of keratinocytes in the normally pigmented epidermis of patients with vitiligo. J. Invest. Dermatol. 1982(79):321-330. 5、 Silvia Moretti, Paolo Fabbri, Gianna Baroni et al. Keratinocyte dysfunction in vitiligo epidermis: cytokine microenvironment and correlation to keratinocyte apoptosis. Histol Histopathol,2009(24): 849-57. 6、 Lee A.Y., Youm Y.H., Kim N.H. et al. Keratinocytes in the depigmented epidermis of vitiligo are more vulnerable to trauma (suction) than keratinocytes in the normally pigmented epidermis, resulting in their apoptosis. Br. J. Dermatol. 2004(151):995-1003. 7、 Lee A.Y., Kim N.H., Choi W.I. Less keratinocyte derived factors related to more keratinocyte apoptosis in depigmented than normally pigmented suction-blistered epidermis may cause passive melanocyte death in vitiligo. J. Invest. Dermatol. 2005(124):976-983. 8、 杜鹃,徐前喜,张建中。寻常型进行期白癜风患者白斑区角质形成细胞凋亡的检测。中华皮肤科杂志,2008,40(1):45-47。 9、 Hockenbery DM, Oltvai ZN, Yin XM,et al.Bcl-2 functions in an antioxidant pathway to prevent apoptosis.Cell,1993,75(2):241-251. |