[1]Lee Y, Ahn C, Han J,et al. The nuclear RNase Ⅲ Drosha initiates microRNA processing.Nature, 2003.425(6956):415-419. [2]He L,Hannon GJ. MicroRNAs:small RNAs with a big role in gene regulation.Nat Rev Genet, 2004.5(7):522-531. [3] Yi R,Qin Y,Macara IG,Cullen BR. Exportin -5 mediates the nuclear export of pre-miRNAs and short hairpin RNAs.Genes Dev, 2003.17(24):3011-3016. [4]Tang G.siRNA and miRNAs :An insight into RISCs.Trends Biochem Sci, 2005.30(2):106-114. [5] Sunkar R ,Girke T,Jain PK,Zhu JK. Cloning and characterization of microRNAs from rice.Plant Cell, 2005.17(5):1397-1411. [6] Kim J,Krichevsky A,et al. Idetification of many microRNAs that copurify with polyribosomes in mammalian neuron.Proc Natl Acad Sci USA, 2004.101(1):360-365. [7] . Lewis BP, Burge CB, Bartel DP. Conserved seed pairing, often flanked by adenosines, indicates that thousands of human genes are microRNA targets. Cell 2005;120:15–20. [PubMed: 15652477] [8] Friedman RC, Farh KK, Burge CB, Bartel DP. Most mammalian mRNAs are conserved targets of microRNAs. Genome Res 2009;19:92–105. [PubMed: 18955434] [9]王震英 徐秀莲 陈楠 ,microRNA与黑素瘤.国际皮肤性病学杂志, 2009年7月 第35卷第4期. [10] Felicetti F,Errico MC,Bottero L,et a1.The promyelocytic leukemia zinc finger-microRNA-221/-222 pathway controls melanoma progression through multiple oncogenic mechanisms.Cancer Res,2008,68(8):2745-2754. [11].Felicetti F,Errico MC,Segnalini P,et a1.MicroRNA-221 and -222 pathway controls melanoma progression.Expert Rev Anti-cancer Ther,2008,8(1 1):1759-1765. [12]Igoucheva O,Alexeev V.MicroRNA-dependent regulation of cKit in cutaneous melanoma.Biochem Biophys Res Commun,2009,379(3):790-794.1] [13].Bemis LT,Chen R,Amato CM,et al.MicroRNA-137 targets microphthalmia-associated transcription factor in melanoma cell lines.Cancer Res.2008.68(5):1362-1368. [14].Lodygin D,Tarasov V,Epanchintsev A,et a1.Inactivation of miR-34a by aberrant CpG methylation in multiple types of cancer.Cell Cycle.2008,7(16):259l-2600. [15] Yan D,Zhou X,Chen X,et a1.MicroRNA-34a inhibits uveal melanoma cell proliferation and migration through downregnlation of c-Met.Invest Ophthalmol Vis Sci.2009.50(4):1559. [16]Migliore C,Petrelli A,Ghiso E,et a1.MicroRNAs impair MET-mediated invasive growth.Cancer Res,2008.68(24):10128-10136. [17] Worley LA,Long MD,Ollken MD,et a1.MicroRNAs associated、with metastasis in uveal melanoma identified by multiplexcd microarray profiling.Melanoma Res.2008.18(3):184-190. [18] Sehultz J,Lorenz P,Gross G,et a1.MicroRNA let-7b targets important celll cycle molecules in malignant melanomacells and interferes with anchomge-independent growth.Cell Res,2008,18(5):549-557. [19] Mailer Dw,Bosserhoff AK integrin beta 3 expression is regufated by let-7a miRNA in malignant melanoma.Oncogene,2008.27(52):6698-6706. [20]. Chen J, Feilotter HE, Zhang X,et al. MicroRNA-193b Represses Cell Proliferationand Regulates Cyclin D1 in Melanoma. Am J Pathol. 2010 Mar 19. [21]Yu J,Ryan DG;,Getsios S,et a1.MicroRNA-184 antagonizes microRNA-205 to maintain SHIP2 levels in epithelia.Proc Natl Acad Sci U S A,2008,105(49):19300-19305. [22]. Sand M, Gambichler T, Skrygan M, et al.Expression Levels of the microRNA Processing Enzymes Drosha and Dicer in Epithelial Skin Cancer. Cancer Invest. 2010 Mar 8. [23]. Skalsky RL, SamolsMA, Plaisance KB. Kaposi’s sarcoma-associated herpesvirus encodes an ortholog of miR-155. J Virol. 2007;81:12836–45. [24] Gottwein E, Cai X, Cullen BR. A novel assay for viral microRNA function identi?es a single nucleotide polymorphism that affects Drosha processing. J Virol 2006;80:5321–6. [25] Tam W, Ben-Yehuda D, Hayward WS. bic, a novel gene activated by proviral insertions in avian leukosis virus-induced lymphomas, is likely to function through its noncoding RNA. Mol Cell Biol 1997;17:1490–1502. [26] Ettehadi P, Greaves MW, Wallach D, Aderka D, Camp RD. Elevated tumour necrosis factor-alpha (TNF-alpha) biological activity in psoriatic skin lesions. Clin Exp Immunol 1994;96:146–151. [27]. Sonkoly E, Wei T, Janson PC, Saaf A, Lundeberg L, Tengvall-Linder M et al. MicroRNAs: novel regulators involved in the pathogenesis of Psoriasis.PLos One ,2007,2 ( 7) :610 - 615 . [28].Dai Y,Huang YS,Tang M.Mieroarray analysis of microRNA expression in peripheral blood cells of systemic lupus erythematosus patients.Lupus,2007.16(12):939-946. [29].李海涛,杨蓉娅,樊昕,[会议论文] 2009 . 2009年京津冀地区皮肤科学术年会论文集. [30].Maurer B, Stanczyk J, Jüngel A,et al. miR-29 is a key regulator of collagen expression in systemic sclerosis. Arthritis Rheum. 2010 Mar 3. [31] 袁晋 傅雯雯 吴文育.斑秃治疗的研究进展. 中国现代医药杂志,2008年11月第 10卷第11期 [32] 蔡晓伟 李 惠.IFN-γ、 IL-10与斑秃关系的初步研究. 华南国防医学杂志,2008年第22卷 第6期 [33] 张西克,高顺强.斑秃与 CTLA-4基因外显子1A/G 49多态性研究.实用心脑肺血管病杂志,2009年 11月第 17卷第 11 期 [34].Goodarzi HR, Abbasi A, Saffari M, et al.MicroRNAs take part in pathophysiology and pathogenesis of Male Pattern Baldness.Mol Biol Rep. 2009 Oct 10. [35] Eberhart JK, He X, Swartz ME, Yan YL, Song H, Boling TC, et al. MicroRNA Mir-140 modulates Pdgf signaling during palatogenesis. Nat Genet 2008;40:290–8. [36].Sun Q, Zhang Y, Yang G, Chen X, Zhang Y, Cao G, et al. Transforming growth factor-beta-regulated miR-24 promotes skeletal muscle differentiation. Nucleic Acids Res 2008;36:2690–9. [37] Taganov KD, Boldin MP, Chang KJ, Baltimore D. NF-kappaB-dependent induction of microRNA miR-146, an inhibitor targeted to signaling proteins of innate immune responses. Proc Natl Acad Sci USA 2006;103:12481–6. [38] Tili E, Michaille JJ, Cimino A, Costinean S, Dumitru CD, Adair B, et al. Modulation of miR-155 and miR-125b levels following lipopolysaccharide/TNF-alpha stimulation and their possible roles in regulating the response to endotoxin shock. J Immunol 2007;179:5082–9. [39].Rozman P.Bolta Z.Use of platelet growth factors in treating wounds and soft-tissue injuries.Acta Dermamvenerol Alp Panonica Adriat.2007.16(4):156-165. [40].Gu J,Iyer VR.P13K signaling and miRNA expression duringthe response of quiescent human fibroblasts to distinct proliferative stimuli.Genome Bio1.2006.7(5):R42. [41].Shilo S.Roy S,Khanna S.MicroRNA in cutaneous wound healing:a new paradigm.DNA Cell Biol,2007,26(4):227. |