[1] |
Clark RA. Resident memory T cells in human health and disease[J]. Sci Transl Med, 2015,7(269):269rv1. doi: 10.1126/scitranslmed. 3010641.<br />
|
[2] |
Cheuk S, Wikén M, Blomqvist L, et al. Epidermal Th22 and Tc17 cells form a localized disease memory in clinically healed psoriasis[J]. J Immunol, 2014,192(7):3111⁃3120. doi: 10.4049/jimmunol. 1302313.<br />
|
[3] |
Suárez⁃Fariñas M, Fuentes⁃Duculan J, Lowes MA, et al. Resolved psoriasis lesions retain of a subset of disease⁃related genes[J]. J Invest Dermatol, 2011,131(2):391⁃400. doi: 10.1038/jid.2010.280.<br />
|
[4] |
Gaffen SL, Jain R, Garg AV, et al. The IL⁃23⁃IL⁃17 immune axis: from mechanisms to therapeutic testing[J]. Nat Rev Immunol, 2014,14(9):585⁃600. doi: 10.1038/nri3707.<br />
|
[5] |
Cai Y, Shen X, Ding C, et al. Pivotal role of dermal IL⁃17⁃producing γδ T cells in skin inflammation[J]. Immunity, 2011,35(4):596⁃610. doi: 10.1016/j.immuni.2011.08.001.<br />
|
[6] |
Ramírez⁃Valle F, Gray EE, Cyster JG. Inflammation induces dermal Vγ4+ γδT17 memory⁃like cells that travel to distant skin and accelerate secondary IL⁃17⁃driven responses[J]. Proc Natl Acad Sci U S A, 2015,112(26):8046⁃8051. doi: 10.1073/pnas. 1508990112.<br />
|
[7] |
Lowes MA, Suárez⁃Fariñas M, Krueger JG. Immunology of psoriasis[J]. Annu Rev Immunol, 2014,32:227⁃255. doi: 10.1146/annurev⁃immunol⁃032713⁃120225.<br />
|
[8] |
Girolomoni G, Mrowietz U, Paul C. Psoriasis: rationale for targeting interleukin⁃17[J]. Br J Dermatol, 2012,167(4):717⁃724. doi: 10.1111/j.1365⁃2133.2012.11099.x.<br />
|
[9] |
Teng MW, Bowman EP, McElwee JJ, et al. IL⁃12 and IL⁃23 cytokines: from discovery to targeted therapies for immune⁃mediated inflammatory diseases[J]. Nat Med, 2015,21(7):719⁃729. doi: 10.1038/nm.3895.<br />
|
[10] |
Gray EE, Suzuki K, Cyster JG. Cutting edge: identification of a motile IL⁃17⁃producing gammadelta T cell population in the dermis[J]. J Immunol, 2011,186(11):6091⁃6095. doi: 10.4049/jimmunol.1100427.<br />
|
[11] |
Park CO, Kupper TS. The emerging role of resident memory T cells in protective immunity and inflammatory disease[J]. Nat Med, 2015,21(7):688⁃697. doi: 10.1038/nm.3883.<br />
|
[12] |
Jiang X, Clark RA, Liu L, et al. Skin infection generates non⁃migratory memory CD8+ T(RM) cells providing global skin⁃immunity[J]. Nature, 2012,483(7388):227⁃231. doi: 10.1038/nature10851.<br />
|
[13] |
Wakim LM, Waithman J, van Rooijen N, et al. Dendritic cell⁃induced memory T cell activation in nonlymphoid tissues[J]. Science, 2008,319(5860):198⁃202. doi: 10.1126/science. 1151869.<br />
|
[14] |
Bhushan M, Bleiker TO, Ballsdon AE, et al. Anti⁃E⁃selectin is ineffective in the treatment of psoriasis: a randomized trial[J]. Br J Dermatol, 2002,146(5):824⁃831.<br />
|
[15] |
Boyman O, Hefti HP, Conrad C, et al. Spontaneous development of psoriasis in a new animal model shows an essential role for resident T cells and tumor necrosis factor⁃alpha[J]. J Exp Med, 2004,199(5):731⁃736. doi: 10.1084/jem.20031482.<br />
|
[16] |
Mackay LK, Rahimpour A, Ma JZ, et al. The developmental pathway for CD103(+)CD8+ tissue⁃resident memory T cells of skin[J]. Nat Immunol, 2013,14(12):1294⁃1301. doi: 10.1038/ni. 2744.<br />
|
[17] |
Mackay LK, Braun A, Macleod BL, et al. Cutting edge: CD69 interference with sphingosine⁃1⁃phosphate receptor function regulates peripheral T cell retention[J]. J Immunol, 2015,194(5):2059⁃2063. doi: 10.4049/jimmunol.1402256.<br />
|
[18] |
Cibrian D, Saiz ML, de la Fuente H, et al. CD69 controls the uptake of L⁃tryptophan through LAT1⁃CD98 and AhR⁃dependent secretion of IL⁃22 in psoriasis [J]. Nat Immunol, 2016,17(8):985⁃96. doi: 10.1038/ni.3504.<br />
|
[19] |
Hartwig T, Pantelyushin S, Croxford AL, et al. Dermal IL⁃17⁃producing γδ T cells establish long⁃lived memory in the skin[J]. Eur J Immunol, 2015,45(11):3022⁃3033. doi: 10.1002/eji. 20154 5883.<br />
|