[1] |
Giunta A, Ventura A, Chimenti MS, et al. Spotlight on ixekizumab for the treatment of moderate⁃to⁃severe plaque psoriasis: design, development, and use in therapy[J]. Drug Des Devel Ther, 2017,11:1643⁃1651. doi: 10.2147/DDDT.S92128.
|
[2] |
Frieder J, Kivelevitch D, Menter A. Calcipotriene betamethasone dipropionate aerosol foam in the treatment of plaque psoriasis: a review of the literature[J]. Ther Deliv, 2017,8(9):737⁃746. doi: 10.4155/tde⁃2017⁃0058.
|
[3] |
Gaspari AA. Innate and adaptive immunity and the pathophysiology of psoriasis[J]. J Am Acad Dermatol, 2006,54(3 Suppl 2):S67⁃S80. doi: 10.1016/j.jaad.2005.10.057.
|
[4] |
Cheuk S, Wikén M, Blomqvist L, et al. Epidermal Th22 and Tc17 cells form a localized disease memory in clinically healed psoriasis[J]. J Immunol, 2014,192(7):3111⁃3120. doi: 10.4049/jimmunol.1302313.
|
[5] |
Grice EA, Kong HH, Conlan S, et al. Topographical and temporal diversity of the human skin microbiome[J]. Science, 2009,324(5931):1190⁃1192. doi: 10.1126/science.1171700.
|
[6] |
Harmsen HJ, de Goffau MC. The human gut microbiota[J]. Adv Exp Med Biol, 2016,902:95⁃108. doi: 10.1007/978⁃3⁃319⁃31248⁃4_7.
|
[7] |
Mayer EA. Gut feelings: the emerging biology of gut⁃brain communication[J]. Nat Rev Neurosci, 2011,12(8):453⁃466. doi: 10.1038/nrn3071.
|
[8] |
Petra AI, Panagiotidou S, Hatziagelaki E, et al. Gut⁃microbiota⁃brain axis and its effect on neuropsychiatric disorders with suspected immune dysregulation[J]. Clin Ther, 2015,37(5):984⁃995. doi: 10.1016/j.clinthera.2015.04.002.
|
[9] |
Jacobs JP, Braun J. Immune and genetic gardening of the intestinal microbiome[J]. FEBS Lett, 2014,588(22):4102⁃4111. doi: 10. 1016/j.febslet.2014.02.052.
|
[10] |
Ley RE, Hamady M, Lozupone C, et al. Evolution of mammals and their gut microbes[J]. Science, 2008,320(5883):1647⁃1651. doi: 10.1126/science.1155725.
|
[11] |
Lin L, Zhang J. Role of intestinal microbiota and metabolites on gut homeostasis and human diseases[J]. BMC Immunol, 2017,18(1):2. doi: 10.1186/s12865⁃016⁃0187⁃3.
|
[12] |
Baker BS, Powles A, Fry L. Peptidoglycan: a major aetiological factor for psoriasis?[J]. Trends Immunol, 2006,27(12):545⁃551. doi: 10.1016/j.it.2006.10.001.
|
[13] |
Fry L, Baker BS, Powles AV, et al. Is chronic plaque psoriasis triggered by microbiota in the skin?[J]. Br J Dermatol, 2013,169(1):47⁃52. doi: 10.1111/bjd.12322.
|
[14] |
Yamasaki K, Di NA, Bardan A, et al. Increased serine protease activity and cathelicidin promotes skin inflammation in rosacea[J]. Nat Med, 2007,13(8):975⁃980. doi: 10.1038/nm1616.
|
[15] |
Morizane S, Gallo RL. Antimicrobial peptides in the pathogenesis of psoriasis[J]. J Dermatol, 2012,39(3):225⁃230. doi: 10.1111/j. 1346⁃8138.2011.01483.x.
|
[16] |
Wang F, Zhang X, Xia P, et al. Enhancement of mRNA of survivin and human beta⁃defensin⁃3 in lesions of psoriasis vulgaris[J]. Eur J Dermatol, 2016,26(1):28⁃33. doi: 10.1684/ejd. 2015.2698.
|
[17] |
Jin T, Sun Z, Chen X, et al. Serum human beta⁃defensin⁃2 is a possible biomarker for monitoring response to JAK inhibitor in psoriasis patients[J]. Dermatology, 2017,233(2⁃3):164⁃169. doi: 10.1159/000475809.
|
[18] |
Ong PY, Ohtake T, Brandt C, et al. Endogenous antimicrobial peptides and skin infections in atopic dermatitis[J]. N Engl J Med, 2002,347(15):1151⁃1160. doi: 10.1056/NEJMoa021481.
|
[19] |
Gao Z, Tseng CH, Strober BE, et al. Substantial alterations of the cutaneous bacterial biota in psoriatic lesions[J/OL]. PLoS One, 2008,3(7):e2719. doi: 10.1371/journal.pone.0002719.
|
[20] |
Zanvit P, Konkel JE, Jiao X, et al. Antibiotics in neonatal life increase murine susceptibility to experimental psoriasis[J]. Nat Commun, 2015,6:8424. doi: 10.1038/ncomms9424.
|
[21] |
Rudramurthy SM, Honnavar P, Chakrabarti A, et al. Association of Malassezia species with psoriatic lesions[J]. Mycoses, 2014,57(8):483⁃488. doi: 10.1111/myc.12186.
|
[22] |
Pietrzak A, Grywalska E, Socha M, et al. Prevalence and possible role of Candida Species in patients with psoriasis: a systematic review and meta⁃analysis[J/OL]. Mediators Inflamm, 2018,2018:9602362. doi: 10.1155/2018/9602362.
|
[23] |
Fry L, Baker BS, Powles AV, et al. Psoriasis is not an autoimmune disease?[J]. Exp Dermatol, 2015,24(4):241⁃244. doi: 10.1111/exd.12572.
|
[24] |
Kim J, Krueger JG. The immunopathogenesis of psoriasis[J]. Dermatol Clin, 2015,33(1):13⁃23. doi: 10.1016/j.det.2014.09.002.
|
[25] |
Mahdavinia M, Rasmussen HE, Engen P, et al. Atopic dermatitis and food sensitization in South African toddlers: role of fiber and gut microbiota[J]. Ann Allergy Asthma Immunol, 2017,118(6):742⁃743.e3. doi: 10.1016/j.anai.2017.04.011.
|
[26] |
Chan CW, Wong RS, Law PT, et al. Environmental factors associated with altered gut microbiota in children with eczema: a systematic review[J]. Int J Mol Sci, 2016,17(7):1147. doi: 10. 3390/ijms17071147.
|
[27] |
Krueger JG. Hiding under the skin: a welcome surprise in psoriasis[J]. Nat Med, 2012,18(12):1750⁃1751. doi: 10.1038/nm.3025.
|
[28] |
Eppinga H, Sperna WCJ, Thio HB, et al. Similar depletion of protective faecalibacterium prausnitzii in psoriasis and inflammatory bowel disease, but not in hidradenitis suppurativa[J]. J Crohns Colitis, 2016,10(9):1067⁃1075. doi: 10.1093/ecco⁃jcc/jjw070.
|
[29] |
Scher JU, Ubeda C, Artacho A, et al. Decreased bacterial diversity characterizes the altered gut microbiota in patients with psoriatic arthritis, resembling dysbiosis in inflammatory bowel disease[J]. Arthritis Rheumatol, 2015,67(1):128⁃139. doi: 10.1002/art.38892.
|