银屑病基因-环境、基因-基因交互作用研究进展

Abstract

SHEN CHANGBING Xuejun Zhang. Gene-environment and gene-gene interactions in psoriasis[J].Chinese Journal of Dermatology, 2014, 47(11): 835-838.

[1]Dempfle A, Scherag A, Hein R, et al.Gene–environment interactions for complex traits: definitions,methodological requirements and challenges[J].Eur J Hum Genet, 2008, 16(10):1164-1172
[2]袁芳, 刘盼盼, 徐进，等.基因-基因环境交互作用分析方法的比较[J].宁波大学学报理工版, 2012, 25(4):115-119
[3]Jin Y, Yang S, Zhang F, et al.Combined effects of HLA-Cw6 and cigarette smoking in psoriasis vulgaris: A hospital-based case–control study in China[J].J Eur Acad Dermatol Venereol, 2009, 23(2):132-137
[4]高敏, 张学军, 魏生才等.等位基因与银屑病环境危险因素的交互作用[J].中国麻风皮肤病杂志, 2003, 19(1):22-24
[5]Zheng GY, Wei SC, Shi TL, et al.Association between alcohol,smoking and HLA-DQA1* 0201 genotype in psoriasis[J].Acta Biochim Biophys Sin, 2004, 36(9):597-602
[6]Yin XY, Cheng H, Wang WJ, et al.TNIP1ANXA6 and CSMD1 Variants Interacting with Cigarette Smoking,Alcohol Intake Affect Risk of Psoriasis[J].J Dermatol Sci, 2013, 70(2):94-98
[7]魏生才, 高敏, 张学军等.银屑病患者的环境因素与 -等位基因关联研究[J].中华流行病学杂志, 2002, 23(6):445-446
[8]Jin Y, Zhang F, Yang S, et al.Combined effects of HLA-Cw6,body mass index and waist–hip ratio on psoriasis vulgaris in Chinese Han population[J].J Dermatol Sci, 2008, 52(2):123-129
[9]Li WQ, Han JL, Zhang MF, et al.Interactions between adiposity and genetic polymorphisms on the risk of psoriasis[J].Br J Dermatol, 2013, 168(3):639-642
[10]E Rácz, D Kurek, M Kant, et al.GATA3 expression is decreased in psoriasis and during epidermal regeneration; induction by narrow-band UVB and IL-4[J].PloS one, 2011, 6(5):e19806-
[11]Hüffmeier U, Lascorz J, Becker T, et al.Characterisation of psoriasis susceptibility locus 6 (PSORS6) in patients with early onset psoriasis and evidence for interaction with PSORS1[J].J Med Genet, 2009, 46(11):736-744
[12]Vasilopoulos Y, Sagoo GS, Cork MJ, et al.HLA-C,CSTA and DS12346 susceptibility alleles confer over 100-fold increased risk of developing psoriasis: evidence of gene interaction[J].J Hum Genet, 2011, 56(6):423-427
[13]Sánchez FO, Linga Reddy MV, Mallbris L, et al.IFN-regulatory factor 5 gene variants interact with the class I MHC locus in the Swedish psoriasis population[J].J Invest Dermatol, 2008, 128(7):1704-1709
[14]Vasilopoulos Y, Walters K, Cork MJ, et al.Association analysis of the skin barrier gene cystatin A at the PSORS5 locus in psoriatic patients: evidence for interaction between PSORS1 and PSORS5[J].Eur J Hum Genet, 2008, 16(8):1002-1009
[15]Strange A, Capon F, Spencer CC, et al.A genome-wide association study identifies new psoriasis susceptibility loci and an interaction between HLA-C and ERAP1[J].Nat genet, 2010, 42(11):985-990
[16]Lysell J, Padyukov L, Kockum I, et al.Genetic Association with ERAP1 in Psoriasis Is Confined to Disease Onset after Puberty and Not Dependent on HLA-C*06[J].J Invest Dermatol, 2013, 133(2):411-417
[17]Zhang XJ, Huang W, Yang S, et al.Psoriasis genome-wide association study identifies susceptibility variants within LCE gene cluster at 1q21[J].Nat genet, 2009, 41(2):205-210
[18]Nair RP, Duffin KC, Helms C, et al.Genome-wide scan reveals association of psoriasis with IL-23 and NF-κB pathways[J].Nat genet, 2009, 41(2):199-204
[19]Zheng HF, Zuo XB, Lu WS, et al.Variants in MHC,LCE and IL12B have epistatic effects on psoriasis risk in Chinese population[J].J Dermatol Sci, 2011, 61(2):124-128
[20]de Cid R, Riveira-Munoz E, Zeeuwen PL, et al.Deletion of the late cornified envelope LCE3B and LCE3C genes as a susceptibility factor for psoriasis[J].Nat genet, 2009, 41(2):211-215
[21]Hüffmeier U, Bergboer JG, Becker T, et al.Replication of LCE3C_LCE3B CNV as a risk factor for psoriasis and analysis of interaction with other genetic risk factors[J].J Invest Dermatol, 2009, 130(4):979-984
[22]Riveira-Munoz E, He SM, Escaramís G, et al.Meta-analysis confirms the LCE3C_LCE3B deletion as a risk factor for psoriasis in several ethnic groups and finds interaction with HLA-Cw6[J].J Invest Dermatol, 2010, 131(5):1105-1109
[23]Xu L, Li Y, Zhang X, et al.Deletion of LCE3C and LCE3B genes is associated with psoriasis in a northern Chinese population[J].Br J Dermatol, 2011, 165(4):882-887
[24]Li M, Wu Y, Chen G, et al.Deletion of the late cornified envelope genes LCE3C and LCE3B is associated with psoriasis in a Chinese population[J].J Invest Dermatol, 2011, 131(8):1639-1643
[25]Ammar M, Bouazizi F, Bouhaha R, et al.Association analysis of LCE3C_LCE3B deletion in Tunisian psoriatic population[J].Arch Dermatol Res, 2012, 304(9):733-738
[26]Wang WJ, Yin XY, Zuo XB, et al.Gene-gene interactions in IL23Th17 pathway contribute to psoriasis susceptibility in Chinese Han population[J].J Eur Acad Dermatol Venereol, 2013, 27(9):1156-1162

Gene-environment and gene-gene interactions in psoriasis

PDF

Like

Knowledge

Abstract

Cite this article

share this article

References

Related Articles 15

Metrics

Comments

Recommended 10

[1]	Chen Yiwen, Su Ting, Su Zhonglan. Association between psoriasis and STAT3 [J]. Chinese Journal of Dermatology, 2019, 52(7): 502-505.
[2]	Su Beibei, Gan Quan, Gan Caibin. Effects of metformin on inflammatory status and phosphorylated Stat3 expression in lesions of a psoriasis?like mouse model [J]. Chinese Journal of Dermatology, 2019, 52(7): 475-480.
[3]	Qin Hui, Zheng Jie. FTY720 ameliorates psoriasis by inhibiting infiltration of skin Interleukin 17-producing γδT cells [J]. Chinese Journal of Dermatology, 2019, 52(6): 395-400.
[4]	Wang Liwei, Luo Lingling, Cui Pan′gen, Li Min. Relationship between intestinal flora and psoriasis [J]. Chinese Journal of Dermatology, 2019, 52(5): 357-360.
[5]	Hu Yu, Chen Min, Gu Heng. MicroRNA and lncRNA in psoriasis [J]. Chinese Journal of Dermatology, 2019, 52(5): 350-353.
[6]	Pan Jiaxu, Zhou Jing. NF?κB signaling pathway in psoriasis [J]. Chinese Journal of Dermatology, 2019, 52(5): 361-363.
[7]	Zeng Jingxin, Tian Xin, Zhu Huilan, Zhang Xibao, Lin Ling, Zhang Lidan, Liu Weiyu, Luo Quan . Expression of miRNA-148a-3p in CD4⁺ T lymphocytes in peripheral blood of patients with psoriasis vulgaris and its clinical significance [J]. Chinese Journal of Dermatology, 2019, 52(4): 231-235.
[8]	Committee on Psoriasis, Chinese Society of Dermatology. Guideline for the diagnosis and treatment of psoriasis in China （2018 simplified edition） [J]. Chinese Journal of Dermatology, 2019, 52(4): 223-230.
[9]	Working Committee on Standardized Diagnosis and Treatment, China Dermatologist Association, Dermatological Surgical Equipment Group and Skin Phototherapy Group, Committee on Skin Disease and Cosmetic Dermatology, China Association of Medical Equipment. Clinical application of home narrow band ultraviolet B phototherapy: an expert consensus statement [J]. Chinese Journal of Dermatology, 2019, 52(3): 156-161.
[10]	Gao Yimeng, Jin Hongzhong. The role of neuropsychiatric factors in the occurrence of psoriasis [J]. Chinese Journal of Dermatology, 2019, 52(3): 204-207.
[11]	Guan Xin, Zhang Chunlei. Evaluation of efficacy of infliximab in the long-term treatment of moderate and severe psoriasis vulgaris [J]. Chinese Journal of Dermatology, 2019, 52(2): 86-89.
[12]	WANG Gang. Main adverse reactions in biological agents in dermatology and their strategies [J]. Chinese Journal of Dermatology, 2019, 52(2): 77-80.
[13]	Hu Yu, Luan Chao, Lian Ni, Hao Zhimin, Sun Yujie, Wang Yan, Gu Heng, Chen Min. Expression of proprotein convertase subtilisin/kexin type 9 in the plasma of patients with psoriasis and its effect on the activation of peripheral CD4+ T cells [J]. Chinese Journal of Dermatology, 2019, 52(2): 90-93.
[14]	Peng Chen, Chen Wenjuan, Yu Ning, Gao Yunlu, Yi Xuemei, Ding Yangfeng. The progress of relationship between psoriasis and microecology of skin and intestine [J]. Chinese Journal of Dermatology, 2019, 52(2): 135-137.
[15]	Xiong Juanjuan, Bi Jianping, Sun Dexiang, Li Nian, Lu Qiaoyun. Dermoscopic features of onychomycosis and nail psoriasis [J]. Chinese Journal of Dermatology, 2019, 52(2): 106-110.