[1] |
苏芳, 晋亮, 刘玮, 等. MicroRNA⁃125a在寻常型银屑病皮损中的表达及其对HaCaT细胞增殖的影响[J]. 中华皮肤科杂志, 2020,53(11):886⁃890. doi: 10.35541/cjd.20200480.
|
[2] |
Chan JR, Blumenschein W, Murphy E, et al. IL⁃23 stimulates epidermal hyperplasia via TNF and IL⁃20R2⁃dependent mechanisms with implications for psoriasis pathogenesis[J]. J Exp Med, 2006,203(12):2577⁃2587. doi: 10.1084/jem.20060244.
|
[3] |
Tu Z, Zhang S, Zhou G, et al. LMO4 is a disease⁃provocative transcription coregulator activated by IL⁃23 in psoriatic keratinocytes[J]. J Invest Dermatol, 2018,138(5):1078⁃1087. doi: 10.1016/j.jid.2017.12.010.
|
[4] |
Taki T, Takeichi T, Sugiura K, et al. Roles of aberrant hemichannel activities due to mutant connexin26 in the pathogenesis of KID syndrome[J]. Sci Rep, 2018,8(1):12824. doi: 10.1038/s41598⁃018⁃30757⁃3.
|
[5] |
Kanda N, Watanabe S. IL⁃12, IL⁃23, and IL⁃27 enhance human beta⁃defensin⁃2 production in human keratinocytes[J]. Eur J Immunol, 2008,38(5):1287⁃1296. doi: 10.1002/eji.200738051.
|
[6] |
Timis TL, Orasan RI. Understanding psoriasis: role of miRNAs[J]. Biomed Rep, 2018,9(5):367⁃374. doi: 10.3892/br.2018.1146.
|
[7] |
Srivastava A, Nikamo P, Lohcharoenkal W, et al. MicroRNA⁃146a suppresses IL⁃17⁃mediated skin inflammation and is genetically associated with psoriasis[J]. J Allergy Clin Immunol, 2017,139(2):550⁃561. doi: 10.1016/j.jaci.2016.07.025.
|
[8] |
Hermann H, Runnel T, Aab A, et al. MiR⁃146b probably assists miRNA⁃146a in the suppression of keratinocyte proliferation and inflammatory responses in psoriasis[J]. J Invest Dermatol, 2017,137(9):1945⁃1954. doi: 10.1016/j.jid.2017.05.012.
|
[9] |
Peng H, Liu Y, Tian J, et al. Decreased expression of microRNA⁃125a⁃3p upregulates interleukin⁃23 receptor in patients with Hashimoto′s thyroiditis[J]. Immunol Res, 2015,62(2):129⁃136. doi: 10.1007/s12026⁃015⁃8643⁃3.
|
[10] |
Pan W, Zhu S, Dai D, et al. MiR⁃125a targets effector programs to stabilize Treg⁃mediated immune homeostasis[J]. Nat Commun, 2015,6:7096. doi: 10.1038/ncomms8096.
|
[11] |
Ge Y, Sun M, Wu W, et al. MicroRNA⁃125a suppresses intestinal mucosal inflammation through targeting ETS⁃1 in patients with inflammatory bowel diseases[J]. J Autoimmun, 2019,101:109⁃120. doi: 10.1016/j.jaut.2019.04.014.
|
[12] |
Gañán⁃Gómez I, Wei Y, Yang H, et al. Overexpression of miR⁃125a in myelodysplastic syndrome CD34+ cells modulates NF⁃κB activation and enhances erythroid differentiation arrest[J/OL]. PLoS One, 2014,9(4):e93404[2020⁃06⁃15]. https://journals.plos.org/plosone/article?id=10.1371/journal.pone.0093404. doi: 10. 1371/journal.pone.0093404.
|
[13] |
Svensson D, Gidlöf O, Turczyńska KM, et al. Inhibition of microRNA⁃125a promotes human endothelial cell proliferation and viability through an antiapoptotic mechanism[J]. J Vasc Res, 2014,51(3):239⁃245. doi: 10.1159/000365551.
|
[14] |
Pei D, Cao J, Qin G, et al. Measurement of circulating miRNA⁃125a exhibits good value in the management of etanercept⁃treated psoriatic patients[J]. J Dermatol, 2020,47(2):140⁃146. doi: 10.1111/1346⁃8138.15157.
|
[15] |
Schön MP, Erpenbeck L. The interleukin⁃23/interleukin⁃17 axis links adaptive and innate immunity in psoriasis[J]. Front Immunol, 2018,9:1323. doi: 10.3389/fimmu.2018.01323.
|
[16] |
Wu Y, Lu Z, Chen Y, et al. Replication of association between interleukin⁃23 receptor (IL⁃23R) and its ligand (IL⁃12B) polymorphisms and psoriasis in the Chinese Han population[J]. Hum Immunol, 2010,71(12):1255⁃1258. doi: 10.1016/j.humimm. 2010.09.014.
|
[17] |
贺晓珍, 黄晓雯, 王婷, 等. 靶向白细胞介素⁃23治疗自身免疫性疾病的研究进展[J]. 国际免疫学杂志, 2018,41(6):670⁃676. doi: 10.3760/cma.j.issn.1673⁃4394.2018.06.016.
|
[18] |
Chen D, Huang X, Lu S, et al. MiRNA⁃125a modulates autophagy of thyroiditis through PI3K/AKT/mTOR signaling pathway[J]. Exp Ther Med, 2019,17(4):2465⁃2472. doi: 10.3892/ etm.2019.7256.
|
[19] |
Woo YR, Cho DH, Park HJ. Molecular mechanisms and management of a cutaneous inflammatory disorder: psoriasis[J]. Int J Mol Sci, 2017,18(12):2684. doi: 10.3390/ijms18122684.
|