[1] |
He Y, Li S, Zhang W, et al. Dysregulated autophagy increased melanocyte sensitivity to H2O2 ⁃induced oxidative stress in vitiligo[J]. Sci Rep, 2017,7:42394. doi: 10.1038/srep42394.
|
[2] |
Yi X, Guo W, Shi Q, et al. SIRT3⁃dependent mitochondrial dynamics remodeling contributes to oxidative stress⁃induced melanocyte degeneration in vitiligo[J]. Theranostics, 2019,9(6):1614⁃1633. doi: 10.7150/thno.30398.
|
[3] |
Isono Y, Furuya M, Kuwahara T, et al. FLCN alteration drives metabolic reprogramming towards nucleotide synthesis and cyst formation in salivary gland[J]. Biochem Biophys Res Commun, 2020,522(4):931⁃938. doi: 10.1016/j.bbrc.2019.11.184.
|
[4] |
Frisoli ML, Harris JE. Vitiligo: mechanistic insights lead to novel treatments[J]. J Allergy Clin Immunol, 2017,140(3):654⁃662. doi: 10.1016/j.jaci.2017.07.011.
|
[5] |
Mitra S, De Sarkar S, Pradhan A, et al. Levels of oxidative damage and proinflammatory cytokines are enhanced in patients with active vitiligo[J]. Free Radic Res, 2017,51(11⁃12):986⁃994. doi: 10.1080/10715762.2017.1402303.
|
[6] |
Wu XG, Xu AE. Successful treatment of vitiligo on the scalp of a 9⁃year⁃old girl using autologous cultured pure melanocyte transplantation[J]. Pediatr Dermatol, 2017,34(1):e22⁃e23. doi: 10.1111/pde.13016.
|
[7] |
Hartman TR, Nicolas E, Klein⁃Szanto A, et al. The role of the Birt⁃Hogg⁃Dubé protein in mTOR activation and renal tumorigenesis[J]. Oncogene, 2009,28(13):1594⁃1604. doi: 10. 1038/onc.2009.14.
|
[8] |
Baba M, Furihata M, Hong SB, et al. Kidney⁃targeted Birt⁃Hogg⁃Dube gene inactivation in a mouse model: Erk1/2 and Akt⁃mTOR activation, cell hyperproliferation, and polycystic kidneys[J]. J Natl Cancer Inst, 2008,100(2):140⁃154. doi: 10.1093/jnci/djm288.
|
[9] |
Yan M, Gingras MC, Dunlop EA, et al. The tumor suppressor folliculin regulates AMPK⁃dependent metabolic transformation[J]. J Clin Invest, 2014,124(6):2640⁃2650. doi: 10.1172/JCI71749.
|
[10] |
Rashighi M, Harris JE. Vitiligo pathogenesis and emerging treatments[J]. Dermatol Clin, 2017,35(2):257⁃265. doi: 10.1016/ j.det.2016.11.014.
|
[11] |
Ge Y, Huang M, Yao YM. Autophagy and proinflammatory cytokines: Interactions and clinical implications[J]. Cytokine Growth Factor Rev, 2018,43:38⁃46. doi: 10.1016/j.cytogfr.2018. 07.001.
|
[12] |
Singh S, Singh U, Pandey SS. Serum concentration of IL⁃6, IL⁃2, TNF⁃α, and IFNγ in vitiligo patients[J]. Indian J Dermatol, 2012,57(1):12⁃14. doi: 10.4103/0019⁃5154.92668.
|
[13] |
Harris JE, Harris TH, Weninger W, et al. A mouse model of vitiligo with focused epidermal depigmentation requires IFN⁃γ for autoreactive CD8+ T⁃cell accumulation in the skin[J]. J Invest Dermatol, 2012,132(7):1869⁃1876. doi: 10.1038/jid.2011. 463.
|
[14] |
Yang L, Yang S, Lei J, et al. Role of chemokines and the corresponding receptors in vitiligo: a pilot study[J]. J Dermatol, 2018,45(1):31⁃38. doi: 10.1111/1346⁃8138.14004.
|