[1]O’Connel RM, Rao DS, Chaudhuri AA, et al.Physiological and pathological roles for microRNAs in the immune system.Nat. Rev. Immunol,2010,10(2):111-122[2]刘丹丹,赫晓燕.微小RNA在皮肤疾病中的调控作用研究进展.中华皮肤科杂志,2010,43(12):892-894[3]Sonkoly E, Janson P, Majuri ML, et al.MiR-155 is overexpressed in patients with atopic dermatitis and modulates T-cell proliferative responses by targeting cytotoxic T lymphocyte-associated antigen 4.J Allergy Clin Immunol,2010,126(3):581-589[4]中华医学会皮肤性病学分会免疫学组.中国特应性皮炎诊断和治疗指南.中华皮肤科杂志,2008,41(11):77-l[5]Kunz B,Oranje AP,Labrèze L, et al.Clinical validation and guidelines for the SCORAD index: consensus report of the European Task Force on Atopic Dermatitis.Dermatology,1997,195(1):10-19[6]马蕾,薛海波,周荣佼,等.特应性皮炎患者外周血调节性T细胞与Th17细胞平衡状态分析.中华皮肤科杂志,2012,45(7):481-484[7]马蕾,薛海波,管秀好,等.特应性皮炎患者外周血CD4+T细胞亚群比例变化的研究.中华微生物学和免疫学杂志,2012,32(4):331-332[8]O'Connell RM, Kahn D, Gibson WS, et al.MicroRNA-155 promotes autoimmune inflammation by enhancing inflammatory T cell development.Immunity,2010,33(4):607-619[9]Zhou H, Huang X, Cui H, et al.miR-155 and its star-form partner miR-155* cooperatively regulate type I interferon production by human plasmacytoid dendritic cells.Blood,2010,116(26):5885-5894[10]Yao R, Ma YL, Liang W, et al.MicroRNA-155 modulates Treg and Th17 cells differentiation and Th17 cell function by targeting SOCS1.PLoS One,2012,7(10):e46082-