XU Zu-sen, ZHOU Ying. The role of interferon inducible protein IFI 16 and JAK-STAT pathway in the pathogenesis of systemic lupus erythematosus[J].Chinese Journal of Dermatology, 2006, 39(9): 524-526.
[1] De Andrea M,Ravera R,Gioia D,et al.The interferon system:an overview.Eur J Paediatr Neurol,2002,6 Suppl A:A41-A46. [2] Geng Y,Choubey D.Differential induction of the 200-family proteins in Daudi Burkitt's lymphoma cells by interferon-alpha.J Biol Regul Homeost Agents,2000,14:263-268. [3] Bombardier C,Gladman DD,Urowitz MB,et al.Derivation of the SLEDAI.A disease activity index for lupus patients.The Committee on Prognosis Studies in SLE.Arthritis Rheum,1992,35:630-640. [4] Wei W,Clarke CJ,Somers GR,et al.Expression of IFI 16 in epithelial cells and lymphoid tissues.Histochem Cell Biol,2003,119:45-54. [5] Dawson MJ,Trapani JA.The interferon-inducible autoantigen,IFI 16:localization to the nucleolus and identification of a DNA-binding domain.Biochem Biophys Res Commun,1995,214:152-162. [6] Seelig HP,Ehrfeld H,Renz M.Interferon-gamma-inducible protein p16.A new target of antinuclear antibodies in patients with systemic lupus erythematosus.Arthritis Rheum,1994,37:1672-1683.