[1] Tsicopoulos A,Hamid Q,Varney V,et al. Preferential messenger RNA expression of Th1 type cells (IFN-gamma+ ,IL-2+) in classical delayed type (tuberculin) hypersensitivity reaction in human skin. J Immunol,1992,148:2058-2061. [2] 刘彦群,赵辨.季节性面部接触性皮炎致病因素及其发病机理的初步探讨.中华皮肤科杂志,1992,25:224-227. [3] 庄丽华,赵辨.43例季节性接触性皮炎皮肤试验研究.中华皮肤科杂志,1996, 29:18-21. [4] 严道金,赵辨.季节性接触性皮炎花粉斑贴实验阳性部位皮肤 IgE检测.中华皮肤科杂志,1998, 31:250-251. [5] Del Prete G, De Carli M, Almerigogna F, et al. Human IL-10 is produced by both type 1 helper (Th1) and type 2 helper (Th2) T cell clones and inhibits their antigen specific proliferation and cytokine production. J Immunol,1993,150:353-360. [6] Wynn TA,Morawetz R,Scharton Kersten T, et al. Analysis of granuloma formation in double cytokine deficient mice reveals a central role for IL-10 in polarizing both T helper cell 1-and T helper cell 2-type cytokine response in vivo. J Immunol,1997,159:5014-5023. |