[1] 李新宇综述.银屑病发病机制的现代概念.国外医学皮肤性病学分册,1998,24:137-140. [2] Nickoloff BJ, Karabin GD, Barker JN, et al. Cellular localization of interleukin-8 and its inducer, tumor necrosis factor-alpha in psoriasis. Am J Pathol, 1991, 138:129-140. [3] Schroder J-M,Gregory H,Young J, et al. Neutrophil-activating proteins in psoriasis. J Invest Dermatol,1992,98:241-247. [4] 张开明,刘梅青,尹兴平,等.银屑病患者血清可溶性肿瘤坏死因子受体水平的检测.中华医学杂志,1998,78:524-525. [5] Chomczynski P, Sacchi N. Single-step method of RNA isolation by guanidinium thiocyanate-phenol-chioroform extration. Anal Biochem, 1987,162:156-159. [6] Okubo Y, Koga M. Peripheral blood monocyte in psoriasis patients overproduce cytokines. J Dermatol Sci,1998,17:223-232. [7] Piguet PF, Grau GE, Vesin C, et al. Evolution of collagen arthritis in mice is arrested by treatment with anti-tumor necrosis factor(TNF),antibody or a recombinant soluble TNF receptor. Immunology,1992,77:510-514. [8] Aderka D, Engelmann H, Maor Y, et al. Stabilization of the bioactivity of tumor necrosis factor by its soluble receptors. J Exp Med, 1992,175:323-329. |