[1] |
Gilchrest BA. Photoaging[J]. J Invest Dermatol, 2013, 133(E1): E2⁃6. DOI: 10.1038/skinbio.2013.176.
|
[2] |
Griffiths CE, Russman AN, Majmudar G, et al. Restoration of collagen formation in photodamaged human skin by tretinoin (retinoic acid)[J]. N Engl J Med, 1993, 329(8): 530⁃535. DOI: 10.1056/NEJM199308193290803.
|
[3] |
Li J, Zhang Y, Kuruba R, et al. Roles of microRNA⁃29a in the antifibrotic effect of farnesoid X receptor in hepatic stellate cells[J]. Mol Pharmacol, 2011, 80(1): 191⁃200. DOI: 10.1124/mol. 110.068247.
|
[4] |
Luna C, Li G, Qiu J, et al. Role of miR⁃29b on the regulation of the extracellular matrix in human trabecular meshwork cells under chronic oxidative stress[J]. Mol Vis, 2009, 15: 2488⁃2497.
|
[5] |
Sekiya Y, Ogawa T, Yoshizato K, et al. Suppression of hepatic stellate cell activation by microRNA⁃29b[J]. Biochem Biophys Res Commun, 2011, 412(1): 74⁃79. DOI: 10.1016/j.bbrc.2011.07. 041.
|
[6] |
Li T, Yan X, Jiang M, et al. The comparison of microRNA profile of the dermis between the young and elderly[J]. J Dermatol Sci, 2016, 82(2): 75⁃83. DOI: 10.1016/j.jdermsci.2016.01.005.
|
[7] |
Deng W, Yuan P, Lai W, et al. A novel KRT5 mutation, p.Lys199Asn, is associated with three subtypes of epidermolysis bullosa simplex phenotypes in a single Chinese family[J]. J Dermatol Sci, 2011, 64(3): 241⁃243. DOI: 10.1016/j.jdermsci. 2011.09.003.
|
[8] |
Fisher GJ, Shao Y, He T, et al. Reduction of fibroblast size/mechanical force down⁃regulates TGF⁃β type II receptor: implications for human skin aging[J]. Aging Cell, 2016, 15(1): 67⁃76. DOI: 10.1111/acel.12410.
|
[9] |
刘晨, 赖维, 许庆芳, 等. 连续长波紫外线照射对人皮肤成纤维细胞胞吞、降解弹性蛋白的影响[J]. 中华皮肤科杂志, 2015, 48(5): 338⁃342. DOI: 10.3760/cma.j.issn.0412⁃4030.2015.05. 010.
|
[10] |
Lamore SD, Wondrak GT. UVA causes dual inactivation of cathepsin B and L underlying lysosomal dysfunction in human dermal fibroblasts[J]. J Photochem Photobiol B, 2013, 123: 1⁃12. DOI: 10.1016/j.jphotobiol.2013.03.007.
|
[11] |
Roderburg C, Urban GW, Bettermann K, et al. Micro⁃RNA profiling reveals a role for miR⁃29 in human and murine liver fibrosis[J]. Hepatology, 2011, 53(1): 209⁃218. DOI: 10.1002/hep.23922.
|
[12] |
Suh EJ, Remillard MY, Legesse⁃Miller A, et al. A microRNA network regulates proliferative timing and extracellular matrix synthesis during cellular quiescence in fibroblasts[J]. Genome Biol, 2012, 13(12): R121. DOI: 10.1186/gb⁃2012⁃13⁃12⁃r121.
|
[13] |
van Rooij E, Sutherland LB, Thatcher JE, et al. Dysregulation of microRNAs after myocardial infarction reveals a role of miR⁃29 in cardiac fibrosis[J]. Proc Natl Acad Sci U S A, 2008, 105(35): 13027⁃13032. DOI: 10.1073/pnas.0805038105.
|
[14] |
Plaisier CL, Pan M, Baliga NS. A miRNA⁃regulatory network explains how dysregulated miRNAs perturb oncogenic processes across diverse cancers[J]. Genome Res, 2012, 22(11): 2302⁃2314. DOI: 10.1101/gr.133991.111.
|
[15] |
Sengupta S, den Boon JA, Chen IH, et al. MicroRNA 29c is down⁃regulated in nasopharyngeal carcinomas, up⁃regulating mRNAs encoding extracellular matrix proteins[J]. Proc Natl Acad Sci U S A, 2008, 105(15): 5874⁃5878. DOI: 10.1073/pnas.0801130105.
|
[16] |
Ugalde AP, Español Y, López⁃Otín C. Micromanaging aging with miRNAs: new messages from the nuclear envelope[J]. Nucleus, 2011, 2(6): 549⁃555. DOI: 10.4161/nucl.2.6.17986.
|
[17] |
Ugalde AP, Ramsay AJ, de la Rosa J, et al. Aging and chronic DNA damage response activate a regulatory pathway involving miR⁃29 and p53[J]. EMBO J, 2011, 30(11): 2219⁃2232. DOI: 10.1038/emboj.2011.124.
|