Chinese Journal of Dermatology ›› 2017, Vol. 50 ›› Issue (2): 138-141.
Previous Articles Next Articles
Received:
2017-01-12
Revised:
2017-01-20
Online:
2017-02-15
Published:
2017-01-24
Contact:
WANG Gang
E-mail:xjwgang@fmmu.edu.cn
Add to citation manager EndNote|Reference Manager|ProCite|BibTeX|RefWorks
[1] | Deng Y, Chang C, Lu Q. The inflammatory response in psoriasis: a comprehensive review[J]. Clin Rev Allergy Immunol, 2016, 50(3): 377⁃389. DOI: 10.1007/s12016⁃016⁃8535⁃x. |
[2] | Sticherling M. Psoriasis and autoimmunity[J]. Autoimmun Rev, 2016, 15(12): 1167⁃1170. DOI: 10.1016/j.autrev.2016.09.004. |
[3] | Grant CR, Liberal R, Mieli⁃Vergani G, et al. Regulatory T⁃cells in autoimmune diseases: challenges, controversies and⁃⁃yet⁃⁃unanswered questions[J]. Autoimmun Rev, 2015, 14(2): 105⁃116. DOI: 10.1016/j.autrev.2014.10.012. |
[4] | Bovenschen HJ, van de Kerkhof PC, van Erp PE, et al. Foxp3+ regulatory T cells of psoriasis patients easily differentiate into IL⁃17A⁃producing cells and are found in lesional skin[J]. J Invest Dermatol, 2011, 131(9): 1853⁃1860. DOI: 10.1038/jid.2011.139. |
[5] | Ganguly D, Haak S, Sisirak V, et al. The role of dendritic cells in autoimmunity[J]. Nat Rev Immunol, 2013, 13(8): 566⁃577. DOI: 10.1038/nri3477. |
[6] | Haniffa M, Gunawan M, Jardine L. Human skin dendritic cells in health and disease[J]. J Dermatol Sci, 2015, 77(2): 85⁃92. DOI: 10.1016/j.jdermsci.2014.08.012. |
[7] | Martini E, Wiken M, Cheuk S, et al. Dynamic changes in resident and infiltrating epidermal dendritic cells in active and resolved psoriasis[J]. J Invest Dermatol, 2016. DOI: 10.1016/j.jid.2016.11.033. |
[8] | Singh TP, Zhang HH, Borek I, et al. Monocyte⁃derived inflam⁃matory Langerhans cells and dermal dendritic cells mediate psoriasis⁃like inflammation[J]. Nat Commun, 2016, 7: 13581. DOI: 10.1038/ncomms13581. |
[9] | Kim J, Krueger JG. Highly effective new treatments for psoriasis target the IL⁃23/Type 17 T cell autoimmune Axis[J]. Annu Rev Med, 2017, 68: 255⁃269. DOI: 10.1146/annurev⁃med⁃042915⁃103905. |
[10] | Brinkmann V, Zychlinsky A. Neutrophil extracellular traps: is immunity the second function of chromatin?[J]. J Cell Biol, 2012, 198(5): 773⁃783. DOI: 10.1083/jcb.201203170. |
[11] | Lin AM, Rubin CJ, Khandpur R, et al. Mast cells and neutrophils release IL⁃17 through extracellular trap formation in psoriasis[J]. J Immunol, 2011, 187(1): 490⁃500. DOI: 10.4049/jimmunol. 1100123. |
[12] | Hu SC, Yu HS, Yen FL, et al. Neutrophil extracellular trap forma⁃tion is increased in psoriasis and induces human β⁃defensin⁃2 production in epidermal keratinocytes[J]. Sci Rep, 2016, 6: 31119. DOI: 10.1038/srep31119. |
[13] | Li N, Yamasaki K, Saito R, et al. Alarmin function of cathelicidin antimicrobial peptide LL37 through IL⁃36γ induction in human epidermal keratinocytes[J]. J Immunol, 2014, 193(10): 5140⁃5148. DOI: 10.4049/jimmunol.1302574. |
[14] | Shao S, Cao T, Jin L, et al. Increased lipocalin⁃2 contributes to the pathogenesis of psoriasis by modulating neutrophil chemotaxis and cytokine secretion[J]. J Invest Dermatol, 2016, 136(7): 1418⁃1428. DOI: 10.1016/j.jid.2016.03.002. |
[15] | Boehncke WH, Schön MP. Psoriasis[J]. Lancet, 2015, 386(9997): 983⁃994. DOI: 10.1016/S0140⁃6736(14)61909⁃7. |
[16] | Krueger JG. An autoimmune "attack" on melanocytes triggers psoriasis and cellular hyperplasia[J]. J Exp Med, 2015, 212(13): 2186. DOI: 10.1084/jem.21213insight3. |
[17] | Harden JL, Krueger JG, Bowcock AM. The immunogenetics of psoriasis: a comprehensive review[J]. J Autoimmun, 2015, 64: 66⁃73. DOI: 10.1016/j.jaut.2015.07.008. |
[18] | Zhou F, Cao H, Zuo X, et al. Deep sequencing of the MHC region in the Chinese population contributes to studies of complex disease[J]. Nat Genet, 2016, 48(7): 740⁃746. DOI: 10.1038/ng.3576. |
[19] | Zhang LJ, Sen GL, Ward NL, et al. Antimicrobial peptide LL37 and MAVS signaling drive interferon⁃β production by epidermal keratinocytes during skin Injury[J]. Immunity, 2016, 45(1): 119⁃130. DOI: 10.1016/j.immuni.2016.06.021. |
[20] | Lande R, Botti E, Jandus C, et al. The antimicrobial peptide LL37 is a T⁃cell autoantigen in psoriasis[J]. Nat Commun, 2014, 5: 5621. DOI: 10.1038/ncomms6621. |
[21] | Arakawa A, Siewert K, Stöhr J, et al. Melanocyte antigen triggers autoimmunity in human psoriasis[J]. J Exp Med, 2015, 212(13): 2203⁃2212. DOI: 10.1084/jem.20151093. |
[22] | Shen Z, Wang G, Fan JY, et al. HLA DR B1*04, *07⁃restricted epitopes on keratin 17 for autoreactive T cells in psoriasis[J]. J Dermatol Sci, 2005, 38(1): 25⁃39. DOI: 10.1016/j.jdermsci.2005. 01.001. |
[23] | Jin L, Wang G. Keratin 17: a critical player in the pathogenesis of psoriasis[J]. Med Res Rev, 2014, 34(2): 438⁃454. DOI: 10.1002/med.21291. |
[24] | Shen Z, Chen L, Liu YF, et al. Altered keratin 17 peptide ligands inhibit in vitro proliferation of keratinocytes and T cells isolated from patients with psoriasis[J]. J Am Acad Dermatol, 2006, 54(6): 992⁃1002. DOI: 10.1016/j.jaad.2006.02.033. |
[25] | Fragoulis GE, Siebert S, McInnes IB. Therapeutic targeting of IL⁃17 and IL⁃23 cytokines in immune⁃mediated diseases[J]. Annu Rev Med, 2016, 67: 337⁃353. DOI: 10.1146/annurev⁃med⁃051914⁃021944. |
[26] | Kimball AB, Papp KA, Wasfi Y, et al. Long⁃term efficacy of ustekinumab in patients with moderate⁃to⁃severe psoriasis treated for up to 5 years in the PHOENIX 1 study[J]. J Eur Acad Dermatol Venereol, 2013, 27(12): 1535⁃1545. DOI: 10.1111/jdv.12046. |
[27] | Valenzuela F, Paul C, Mallbris L, et al. Tofacitinib versus etaner⁃cept or placebo in patients with moderate to severe chronic plaque psoriasis: patient⁃reported outcomes from a Phase 3 study[J]. J Eur Acad Dermatol Venereol, 2016, 30(10): 1753⁃1759. DOI: 10.1111/jdv.13702. |
[28] | Papp KA, Bissonnette R, Gooderham M, et al. Treatment of plaque psoriasis with an ointment formulation of the Janus kinase inhibitor, tofacitinib: a Phase 2b randomized clinical trial[J]. BMC Dermatol, 2016, 16(1): 15. DOI: 10.1186/s12895⁃016⁃0051⁃4. |
[29] | Del RJQ, Kircik L. Oral apremilast for the treatment of plaque psoriasis[J]. J Clin Aesthet Dermatol, 2016, 9(9): 43⁃48. |
[30] | Papp KA, Menter MA, Raman M, et al. A randomized phase 2b trial of baricitinib, an oral Janus kinase (JAK) 1/JAK2 inhibitor, in patients with moderate⁃to⁃severe psoriasis[J]. Br J Dermatol, 2016, 174(6): 1266⁃1276. DOI: 10.1111/bjd.14403. |
[31] | Greb JE, Goldminz AM, Elder JT, et al. Psoriasis[J]. Nat Rev Dis Primers, 2016, 2: 16082. DOI: 10.1038/nrdp.2016.82. |
[32] | Linares⁃Pineda TM, Cañadas⁃Garre M, Sánchez⁃Pozo A, et al. Gene polymorphisms as predictors of response to biological therapies in psoriasis patients[J]. Pharmacol Res, 2016, 113(Pt A): 71⁃80. DOI: 10.1016/j.phrs.2016.07.020. |
[33] | van den Reek JM, Coenen MJ, van de L'Isle Arias M, et al. Polymorphisms in CD84, IL12B and TNFAIP3 are associated with response to biologics in patients with psoriasis[J]. Br J Dermatol, 2016. DOI: 10.1111/bjd.15005. |
[34] | Białecka M, Ostasz R, Kurzawski M, et al. IL17A and IL17F gene polymorphism association with psoriasis risk and response to treatment in a polish population[J]. Dermatology, 2016, 232(5): 592⁃596. DOI: 10.1159/000448090. |
[35] | Gallo E, Cabaleiro T, Román M, et al. The relationship between tumour necrosis factor (TNF)⁃α promoter and IL12B/IL⁃23R genes polymorphisms and the efficacy of anti⁃TNF⁃α therapy in psoriasis: a case⁃control study[J]. Br J Dermatol, 2013, 169(4): 819⁃829. DOI: 10.1111/bjd.12425. |
No related articles found! |
|