[1] 李志强, 高天文, 马翠玲, 等. 白癜风患者血清中抗黑素细胞 IgG、IgM抗体的检测及补体介导的血清细胞毒反应. 临床皮肤科杂志, 2000,29:136-138. [2] Munger K, Phelps WC, Bubb V, et al. The E6 and E7 genes of the human papillomavirus type 16 together are necessary and sufficient for transformation of primary human kera tinocytes. J Virol, 1989,63:4417-4421. [3] Halbert CL, Demers GW, Galloway DA. The E7 gene of human papillomavirus type 16 is sufficient for immortalization of human epithe lial cells.J Virol, 1991,65:473-478. [4] 廖文俊综述. HPV E6、E7基因在角朊细胞转化过程中的作用. 国外医学皮肤性病学分册, 1997,23:275-278. [5] Kiyono T, Foster SA, Koop JI, et al.Both Rb/p16INK4a inactivation and telomerase activity are required to immortalize human epi thelial cells.Nature, 1998,396:84-88. [6] Le Poole IC, van den Berg FM, van den Wijngaard RM, et al. Generation of a human melanocyte cell line by introduction of HPV16 E6 and E7 genes. In Vitro Cell Dev Biol Anim, 1997,33:42-49. [7] Le Poole IC, Boissy RE, Sarangarajan R, et al. PIG3V, an immortalized human vitiligo melanocyte cell line, expresses dilated endoplasmic reticulum.In Vitro Cell Dev Biol Anim, 2000,36:309-319. [8] Sun Y, Shah KV, Muller M, et al. Comparison of peptide enzyme-linked immuno sorbent assay and radioimmunoprecipitation assay with in vitro-translated proteins for detection of serum antibodies to human papillomavirus type 16 E6 and E7 proteins. J Clin Microbiol,1994,32:2216-2220. [9] Hoppe-Seyler F, Butz K. Repression of endogenous p53 transactivation function in HeLa cervical carcinoma cells by human papillomavirus type 16 E6, humanmdm-2, and mutant p53. J Virol, 1993,67:3111-3117. |